Bibliography
Found 11 results
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BACE1 Mediates HIV-Associated and Excitotoxic Neuronal Damage Through an APP-Dependent Mechanism. J Neurosci. 2018 ;38(18):4288-4300.
. Altered Oligodendrocyte Maturation and Myelin Maintenance: The Role of Antiretrovirals in HIV-Associated Neurocognitive Disorders. [Internet]. 2015 ;74(11):1093 - 1118. Available from: http://dx.doi.org/10.1097/NEN.0000000000000255
E2F1 in neurons is cleaved by calpain in an NMDA receptor-dependent manner in a model of HIV-induced neurotoxicity. J Neurochem. 2015 ;132(6):742-55.
. Heme oxygenase-1 deficiency accompanies neuropathogenesis of HIV-associated neurocognitive disorders. J Clin Invest. 2014 ;124(10):4459-72.
. Calpain-mediated degradation of MDMx/MDM4 contributes to HIV-induced neuronal damage. Molecular and Cellular Neurosciences [Internet]. 2013 ;57:54-62. Available from: http://www.ncbi.nlm.nih.gov/pubmed/24128662
. Activation status of integrated stress response pathways in neurones and astrocytes of HIV-associated neurocognitive disorders (HAND) cortex. Neuropathology and Applied Neurobiology [Internet]. 2012 ;38(2):175-200. Available from: http://www.ncbi.nlm.nih.gov/pubmed/21883374
. Site-specific hyperphosphorylation of pRb in HIV-induced neurotoxicity. Molecular and Cellular Neurosciences [Internet]. 2011 ;47(2):154-165. Available from: http://www.ncbi.nlm.nih.gov/pubmed/21504794
. E2F1 localizes predominantly to neuronal cytoplasm and fails to induce expression of its transcriptional targets in human immunodeficiency virus-induced neuronal damage. Neuroscience letters [Internet]. 2010 ;479:97-101. Available from: http://www.ncbi.nlm.nih.gov/pubmed/20580656
. Activation of cyclin-dependent kinase 5 by calpains contributes to human immunodeficiency virus-induced neurotoxicity. J Neurochem. 2007 ;103(2):439-55.
Expression of the endoplasmic reticulum stress response marker, BiP, in the central nervous system of HIV-positive individuals. Neuropathol Appl Neurobiol. [Internet]. 2007 ;33:11. Available from: http://www.ncbi.nlm.nih.gov/pubmed/17931354
. Cell cycle proteins exhibit altered expression patterns in lentiviral-associated encephalitis. The Journal of Neuroscience: The Official Journal of the Society for Neuroscience [Internet]. 2002 ;22(6):2185-95. Available from: http://www.ncbi.nlm.nih.gov/pubmed/11896158
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