Bibliography
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Brain inflammation is a common feature of HIV-infected patients without HIV encephalitis or productive brain infection. Curr HIV Res. 2014 ;12(2):97-110.
. Brain large artery inflammation associated with HIV and large artery remodeling. AIDS. 2016 ;30(3):415-23.
. Caspase-1 Activation Is Related With HIV-Associated Atherosclerosis in an HIV Transgenic Mouse Model and HIV Patient Cohort. Arterioscler Thromb Vasc Biol. 2019 ;39(9):1762-1775.
Elevated cerebrospinal fluid Galectin-9 is associated with central nervous system immune activation and poor cognitive performance in older HIV-infected individuals. J Neurovirol. 2019 ;25(2):150-161.
Exosome markers associated with immune activation and oxidative stress in HIV patients on antiretroviral therapy. Sci Rep. 2018 ;8(1):7227.
. Heme oxygenase-1 promoter region (GT)n polymorphism associates with increased neuroimmune activation and risk for encephalitis in HIV infection. J Neuroinflammation. 2018 ;15(1):70.
. JAM-A and ALCAM are therapeutic targets to inhibit diapedesis across the BBB of CD14+CD16+ monocytes in HIV-infected individuals. J Leukoc Biol [Internet]. 2015 ;97(2):401-12. Available from: https://www.ncbi.nlm.nih.gov/pubmed/25420915
. Mononuclear phagocyte accumulation in visceral tissue in HIV encephalitis: evidence for increased monocyte/macrophage trafficking and altered differentiation. Curr HIV Res. 2014 ;12(3):201-12.
. Neurocognitive and neuroinflammatory correlates of PDYN and OPRK1 mRNA expression in the anterior cingulate in postmortem brain of HIV-infected subjects. J Neuroinflammation. 2014 ;11:5.
. Plasma soluble CD163 is associated with postmortem brain pathology in human immunodeficiency virus infection. AIDS. 2017 ;31(7):973-979.
When do models of NeuroAIDS faithfully imitate "the real thing"?. J Neurovirol. 2018 ;24(2):146-155.
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