Bibliography
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HIV-1 Tat induces unfolded protein response and endoplasmic reticulum stress in astrocytes and causes neurotoxicity through glial fibrillary acidic protein (GFAP) activation and aggregation. Journal of Biological Chemistry. 2016 ;291(43):22819-22829.
. HIV-1 Tat alters neuronal autophagy by modulating autophagosome fusion to the lysosome: implications for HIV-associated neurocognitive disorders. J Neurosci. 2015 ;35(5):1921-38.
Mechanisms of HIV-1 Tat neurotoxicity via CDK5 translocation and hyper-activation: role in HIV-associated neurocognitive disorders. Curr HIV Res. 2015 ;13(1):43-54.
. HIV-1 Tat promotes neuronal dysfunction through disruption of microRNAs. Journal of Biological Chemistry [Internet]. 2011 ;286(47):41125-41134. Available from: http://www.ncbi.nlm.nih.gov/pubmed/21956116
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