Bibliography
] HIV alters neuronal mitochondrial fission/fusion in the brain during HIV-associated neurocognitive disorders. Neurobiol Dis. 2016 ;86:154-69.
The HIV Protein gp120 Alters Mitochondrial Dynamics in Neurons. Neurotox Res. 2016 ;29(4):583-93.
HIV-1 Tat alters neuronal autophagy by modulating autophagosome fusion to the lysosome: implications for HIV-associated neurocognitive disorders. J Neurosci. 2015 ;35(5):1921-38.
. Mechanisms of HIV-1 Tat neurotoxicity via CDK5 translocation and hyper-activation: role in HIV-associated neurocognitive disorders. Curr HIV Res. 2015 ;13(1):43-54.
. Role of neurotrophic factor alterations in the neurodegenerative process in HIV associated neurocognitive disorders. Journal of Neuroimmune Pharmacology [Internet]. 2014 ;9(2):102-16. Available from: http://www.ncbi.nlm.nih.gov/pubmed/24510686
Age-dependent molecular alterations in the autophagy pathway in HIVE patients and in a gp120 tg mouse model: reversal with beclin-1 gene transfer. Journal of NeuroVirology [Internet]. 2013 ;19(1):89-101. Available from: http://www.ncbi.nlm.nih.gov/pubmed/23341224
. Alterations in the levels of vesicular trafficking proteins involved in HIV replication in the brains and CSF of patients with HIV-associated neurocognitive disorders. Journal of Neuroimmune Pharmacology [Internet]. 2013 ;8(5):1197-1209. Available from: http://www.ncbi.nlm.nih.gov/pubmed/24292993
. CCAAT/enhancer binding protein β expression is increased in the brain during HIV-1-infection and contributes to regulation of astrocyte tissue inhibitor of metalloproteinase-1. Journal of Neurochemistry [Internet]. 2011 ;118(1):93-104. Available from: http://www.ncbi.nlm.nih.gov/pubmed/21281310
